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全超 博士


学习经历:

2012—2018年 南京大学模式动物研究所  陈帅课题


毕业去向:

美国国立卫生研究院(NIH)  博士后


在所科研成果:

1.Life Metabolism

Impaired SERCA2a phosphorylation causes diabetic cardiomyopathy through impinging on cardiac contractility and precursor protein processing.

2. Nature Communication

    A PKB-SPEG signaling nexus links insulin resistance with diabetic cardiomyopathy by regulating calcium homeostasis.

3. Circulation Research

    SPEG controls calcium re-uptake into the sarcoplasmic reticulum through regulating SERCA2a by its second kinase-domain.

4. Plos One

PKB-mediated Thr649 phosphorylation of AS160/TBC1D4 regulates the R-wave amplitude in the heart.





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